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Other possible mechanisms of anthracycline cardiotoxicity Other mechanisms of cardiotoxicity include direct DNA damage, disruption of the sarcomere protein titin (involved in force regulation of sarcomeres), and alterations in phospholipid content ( 1.1.2
Along with diminished GSH, T2DM patients have an increase in levels of proinflammatory cytokines and MDA, a by-product of lipid peroxidation and measurement for oxidative stress ( in vitro with M
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